miR‑101a‑3p overexpression prevents acetylcholine‑CaCl2‑induced atrial fibrillation in rats via reduction of atrial tissue fibrosis, involving inhibition of EZH2
Improved Binding Affinity and Pharmacokinetics Enable Sustained Degradation of BCL6 In Vivo | Journal of Medicinal Chemistry
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Selective Inhibitors of Protein Methyltransferases | Journal of Medicinal Chemistry
Recombinant Human EGF Protein, CF 236-EG-200: R&D Systems
Biomedicines | Free Full-Text | In Vitro Miniaturized Tuberculosis Spheroid Model | HTML
Organocatalytic Enantioselective Conia‐Ene‐Type Carbocyclization of Ynamide Cyclohexanones: Regiodivergent Synthesis of Morphans and Normorphans - Xu - 2019 - Angewandte Chemie - Wiley Online Library
Cell-Intrinsic and Extrinsic Effects of Galectin-1 and Galectin-3 in B-Cell Precursor Acute Lymphoblastic Leukemia | bioRxiv
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miR‑101a‑3p overexpression prevents acetylcholine‑CaCl2‑induced atrial fibrillation in rats via reduction of atrial tissue fibrosis, involving inhibition of EZH2
PDF) Metabolomic Profiling Reveals Cellular Reprogramming of B-Cell Lymphoma by a Lysine Deacetylase Inhibitor through the Choline Pathway
Requirement for cyclin D3 in germinal center formation and function | Cell Research
miR‑101a‑3p overexpression prevents acetylcholine‑CaCl2‑induced atrial fibrillation in rats via reduction of atrial tissue fibrosis, involving inhibition of EZH2
Thermoresponsive Inverted Colloidal Crystal Hydrogel Scaffolds for Lymphoid Tissue Engineering - Kwak - 2020 - Advanced Healthcare Materials - Wiley Online Library
Discovery of the Clinical Candidate MAK683: An EED-Directed, Allosteric, and Selective PRC2 Inhibitor for the Treatment of Advanced Malignancies | Journal of Medicinal Chemistry
Host CARD11 Inhibits Newcastle Disease Virus Replication by Suppressing Viral Polymerase Activity in Neurons | Journal of Virology
Bcl6 Expressing Follicular Helper CD4 T Cells Are Fate Committed Early and Have the Capacity To Form Memory | The Journal of Immunology
Recombinant Human EGF Protein, CF 236-EG-200: R&D Systems
Identification of the Aryl Hydrocarbon Receptor Target Gene TiPARP as a Mediator of Suppression of Hepatic Gluconeogenesis by 2,3,7,8-Tetrachlorodibenzo-p-dioxin and of Nicotinamide as a Corrective Agent for This Effect* - Journal of
Alternative splicing of METTL3 explains apparently METTL3-independent m6A modifications in mRNA | PLOS Biology
